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Zack
"OCSyndrome(OCD) = Defaulted Gene = { Glutamate Transporter Gene= 'SLC1A1' } in the condition labeled OCD. The job of the glutamate transporter gene is to remove excess glutamate form the Nerve Cell into the Glial Cell. Every person that has been diagnosed with OCD & had a [glutamate level in the brain test] has tested possitive for excesive levels of glutamate in their brains. Excessive glutamate has been proven to affect short term memory just like people with ALS. People with OCSnyndrome have adequate glutamate transporters except the glutamate transporters are not able to remove excess glutamate adequately. People with the condition labeled OCD have bad short term memory. Taurine can be substituted for glutamate. Taurine will activate the glutamate receptor without over exciting the cell nor over ecxiting the cell to death. "
My URL: http://www.ocdtribe.com/Zack

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From: luna_winter
August 30, 2010, 10:54 am
I thought it was something like math or science. I'm horrible at math so I wouldn't be able to program anything.

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'Have no fear of perfection, you'll never reach it.' Salvador Dali
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From: luna_winter
August 27, 2010, 11:50 am
Thanks! I like your page to. I read your occupation as student, what are you studying?

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'Have no fear of perfection, you'll never reach it.' Salvador Dali
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From: AshleeNic
July 19, 2010, 4:52 pm
ur awesome Zack! hope all is well in your neck of the woods ;)

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'More than that, we rejoice in our sufferings, knowing that suffering produces endurance, and endurance produces character, and character produces hope' Romans 5:3-4
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From: Zack
July 3, 2010, 11:57 am
WHAT IS THE CONDITION LABELED OCD ?


The mislabeled condition OCD is caused by ones inability to remove excess GLUTAMIC ACID/glutamate adequately from the glutamate receptor N-methyl D-aspartate resulting in excessive and toxic levels of the most common excitory neurotransmitter GLUTAMIC ACID/glutamate. When excessive levels of glutamate activate the NMDAR (N-methyl D-aspartate receptor) this causes excitoxicity via increasing Choline and decreasing Myo-Inositol.

Inadequate removal of Excess glutamate from the glutamate receptor N-methyl D-aspartate causes excitotoxicity via Choline increase and Myo-Inositol decrease inducing Obsessive Compulsive Syndrome.

That means that the mislabeled condition OCD is induced by increased levels of Choline and decreased levels of Myo-Inositol. That means the mislabeled condition OCD is caused by NMDAR (N-methyl D-aspartate Receptor) excitotoxicity activated (triggered) by excessive levels of glutamate. Yes! Your condition labeled OCD is caused by actual excitotoxicity activated by excess glutamate within your brain. Excitotoxicity causes cell death. NMDAR Excitotoxicity cell death is induced by the increased level of Choline triggered by excess glutamate within the NMDAR (N-methyl D-aspartate Receptor). Excitotoxicity is also associated with Alzheimer's disease. That makes anyone with the condition labeled OCD a prime candidate for Alzheimer's disease.

An appropriate name is:
Excess Glutamate Activated N-methyl D-aspartate Receptor Increasing Choline Inducing Excitotoxicity Disease
EGANMDARICIED



OC (OCD) is caused by ones inability to process GLUTAMIC ACID/glutamate correctly resulting in excessive and toxic levels of the most common excitory neurotransmitter GLUTAMIC ACID/glutamate. GLUTAMIC ACID/glutamate functions as the most common excitatory neurotransmitter in the brain and spinal cord. GLUTAMIC ACID/glutamate is non-essential amino acid; commonly found in most foods. Some foods contain higher levels then others. The effect is a over stimulated brain, the over stimulated analytical and habit learning processors of the brain is what causes OC (OCD). It retards short term memory. This causes one to easily forget names of people if not recited or if it is not associated with a symbolism. A symbolism for EXAMPLE: Memorising phone # 365-1175 There is 365 days in a year, one short of a dozen is 11, A quarter short of 100 is 75. Toxic levels of GLUTAMIC ACID/glutamate causes healthy cells to die. Obsessive compulsive is a symptom of the over stimulated analytical and habit learning processors of the brain. OC is not a disorder itself. That means OC is a symptom and not a disorder. The actual disorder is ones inability to process GLUTAMIC ACID/glutamate correctly resulting in excessive and toxic levels of the most common excitatory neurotransmitter GLUTAMIC ACID/glutamate. A non-essential amino acid is a amino acid that is manufactured (PRODUCED) by ones own body; Such as GLUTAMIC ACID/glutamate. Excessive and toxic levels of the most common excitatory neurotransmitter GLUTAMIC ACID/glutamate affects different people's personalities differently; just like drugs. Some people will pull there hair out, some will pick there skin, some will hoard, some will count endlessly, and so on. OCD is currently mislabeled.

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OCSyndrome(OCD) = Defaulted Gene = { Glutamate Transporter Gene= 'SLC1A1' } in the condition labeled OCD. The job of the glutamate transporter gene is to remove excess glutamate form the Nerve Cell into the Glial Cell. Every person that has been diagnosed with OCD & had a [glutamate level in the brain test] has tested possitive for excesive levels of glutamate in their brains. Excessive glutamate has been proven to affect short term memory just like people with ALS. People with OCSnyndrome have adequate glutamate transporters except the glutamate transporters are not able to remove excess glutamate adequately. People with the condition labeled OCD have bad short term memory. Taurine can be substituted for glutamate. Taurine will activate the glutamate receptor without over exciting the cell nor over ecxiting the cell to death.
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From: empleh
July 2, 2010, 12:21 pm
Just when I thought OCD couldn't get more complex. Where did you learn about of all this scientific links to OCD. Great information but I would guess most people done know their glutamate from their Glial cell against their ALS or Myo-inositol ??..WTF ??.. but full respect for putting the info there.

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